{"id":133933,"date":"1997-01-01T00:00:00","date_gmt":"1997-01-01T00:00:00","guid":{"rendered":"https:\/\/www.deberes.net\/tesis\/sin-categoria\/reaccion-biologica-proteica-en-el-infarto-agudo-de-miocardio\/"},"modified":"1997-01-01T00:00:00","modified_gmt":"1997-01-01T00:00:00","slug":"reaccion-biologica-proteica-en-el-infarto-agudo-de-miocardio","status":"publish","type":"post","link":"https:\/\/www.deberes.net\/tesis\/ciencias-medicas\/reaccion-biologica-proteica-en-el-infarto-agudo-de-miocardio\/","title":{"rendered":"Reaccion biologica proteica en el infarto agudo de miocardio."},"content":{"rendered":"<h2>Tesis doctoral de <strong>  Sanchez Salvador Jos\u00e9 Tomas <\/strong><\/h2>\n<p>En el cuadro clinico del iam predomina un sintoma general, el dolor y se acompa\u00f1a de un sindrome biologico caracterizado por fiebre, leucocitosis, aumento de la vsg y aumento en la concentracion de determinadas proteinas plasmaticas. Estas proteinas reactantes de fase aguda, aparecen como-respuesta al estimulo inflamatorio (iam), siendo liberadas por los hepacitos humanos y su sintesis controlada por las citokinas.  analizadas evolutivamente el curso del iam, presentan uniformidad de resultados para las proteinas de migraci-on alfa-1 (a1ga y a1at) y alfa-2 (hp), con asincronia en la evolucion para la pcr (proteina de migracion beta). Observandose un ascenso precoz de las proteinas de fase aguda, incluso antes que las enzimas de necrosis miocardica ck y ckmb, dato no comunicado en la literatura; y permanecia de concentraciones elevadas de las proteinas reactantes, al menos durante 8-10 dias.  a1ga: alfa 1 glicoproteina acida; a1at: alfa 1 antitripsina. Hp: haptoglobina; pcr: proteina c-reactiva.<\/p>\n<p>&nbsp;<\/p>\n<h3>Datos acad\u00e9micos de la tesis doctoral \u00ab<strong>Reaccion biologica proteica en el infarto agudo de miocardio.<\/strong>\u00ab<\/h3>\n<ul>\n<li><strong>T\u00edtulo de la tesis:<\/strong>\u00a0 Reaccion biologica proteica en el infarto agudo de miocardio. <\/li>\n<li><strong>Autor:<\/strong>\u00a0  Sanchez Salvador Jos\u00e9 Tomas <\/li>\n<li><strong>Universidad:<\/strong>\u00a0 Granada<\/li>\n<li><strong>Fecha de lectura de la tesis:<\/strong>\u00a0 01\/01\/1997<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n<h3>Direcci\u00f3n y tribunal<\/h3>\n<ul>\n<li><strong>Director de la tesis<\/strong>\n<ul>\n<li>Antonio Rodr\u00edguez Cuartero<\/li>\n<\/ul>\n<\/li>\n<li><strong>Tribunal<\/strong>\n<ul>\n<li>Presidente del tribunal: Jos\u00e9 Antonio Gomez Capilla <\/li>\n<li>Jos\u00e9 Ram\u00f3n Vicente Rull (vocal)<\/li>\n<li> Candel Delgado Jos\u00e9 Miguel (vocal)<\/li>\n<li>Manuel Martin Gonzalez (vocal)<\/li>\n<\/ul>\n<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n","protected":false},"excerpt":{"rendered":"<p>Tesis doctoral de Sanchez Salvador Jos\u00e9 Tomas En el cuadro clinico del iam predomina un sintoma general, el dolor y [&hellip;]<\/p>\n","protected":false},"author":1,"featured_media":0,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"site-sidebar-layout":"default","site-content-layout":"","ast-site-content-layout":"","site-content-style":"default","site-sidebar-style":"default","ast-global-header-display":"","ast-banner-title-visibility":"","ast-main-header-display":"","ast-hfb-above-header-display":"","ast-hfb-below-header-display":"","ast-hfb-mobile-header-display":"","site-post-title":"","ast-breadcrumbs-content":"","ast-featured-img":"","footer-sml-layout":"","theme-transparent-header-meta":"","adv-header-id-meta":"","stick-header-meta":"","header-above-stick-meta":"","header-main-stick-meta":"","header-below-stick-meta":"","astra-migrate-meta-layouts":"default","ast-page-background-enabled":"default","ast-page-background-meta":{"desktop":{"background-color":"var(--ast-global-color-4)","background-image":"","background-repeat":"repeat","background-position":"center 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