{"id":135949,"date":"1997-01-01T00:00:00","date_gmt":"1997-01-01T00:00:00","guid":{"rendered":"https:\/\/www.deberes.net\/tesis\/sin-categoria\/desarrollo-de-oligonucleotidos-antisentido-bcl-2-liposomiales-para-el-tratamiento-de-linfomas-foliculares\/"},"modified":"1997-01-01T00:00:00","modified_gmt":"1997-01-01T00:00:00","slug":"desarrollo-de-oligonucleotidos-antisentido-bcl-2-liposomiales-para-el-tratamiento-de-linfomas-foliculares","status":"publish","type":"post","link":"https:\/\/www.deberes.net\/tesis\/ciencias-medicas\/desarrollo-de-oligonucleotidos-antisentido-bcl-2-liposomiales-para-el-tratamiento-de-linfomas-foliculares\/","title":{"rendered":"Desarrollo de oligonucleotidos antisentido bcl-2 liposomiales para el tratamiento de linfomas foliculares."},"content":{"rendered":"<h2>Tesis doctoral de <strong> Mar Tormo Diaz <\/strong><\/h2>\n<p>El 90% de los linfomas foliculares tienen la traslocacion t(14;18), y como consecuencia del gen bcl-2, que actua inhibiendo las apoptosis, se encuentra sobreexpresado.  oligonucleotidos antisentido bcl-2 incorporados en liposomas han demostrado que son capaces de inhibir el crecimiento tumoral de una linea celular de linfoma folicular. Ademas, estos oligonucleotidos inhibieron especialmente la proteina bcl-2 en estas celulas, asi como, la observada en otra linea celular tumoral linfoide de otra estirpe (leucemia t). La inhibicion del crecimiento celular observada en las celulas de linfoma folicular es mediada por la induccion de la apoptosis.  ademas, este efecto parece ser consecuencia de una disminucion de la relacion existente entre las proteinas bcl-2\/bax.<\/p>\n<p>&nbsp;<\/p>\n<h3>Datos acad\u00e9micos de la tesis doctoral \u00ab<strong>Desarrollo de oligonucleotidos antisentido bcl-2 liposomiales para el tratamiento de linfomas foliculares.<\/strong>\u00ab<\/h3>\n<ul>\n<li><strong>T\u00edtulo de la tesis:<\/strong>\u00a0 Desarrollo de oligonucleotidos antisentido bcl-2 liposomiales para el tratamiento de linfomas foliculares. <\/li>\n<li><strong>Autor:<\/strong>\u00a0 Mar Tormo Diaz <\/li>\n<li><strong>Universidad:<\/strong>\u00a0 Universitat de val\u00e9ncia (estudi general)<\/li>\n<li><strong>Fecha de lectura de la tesis:<\/strong>\u00a0 01\/01\/1997<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n<h3>Direcci\u00f3n y tribunal<\/h3>\n<ul>\n<li><strong>Director de la tesis<\/strong>\n<ul>\n<li> Garc\u00eda Conde Bru Javier<\/li>\n<\/ul>\n<\/li>\n<li><strong>Tribunal<\/strong>\n<ul>\n<li>Presidente del tribunal: Vicente Vicente Garcia <\/li>\n<li>Alberto Gra\u00f1ena Batista (vocal)<\/li>\n<li>Salvador Francisco Ali\u00f1o Pellicer (vocal)<\/li>\n<li>Andr\u00e9s Cervantes Ruiperez (vocal)<\/li>\n<\/ul>\n<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n","protected":false},"excerpt":{"rendered":"<p>Tesis doctoral de Mar Tormo Diaz El 90% de los linfomas foliculares tienen la traslocacion t(14;18), y como consecuencia del [&hellip;]<\/p>\n","protected":false},"author":1,"featured_media":0,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"site-sidebar-layout":"default","site-content-layout":"","ast-site-content-layout":"","site-content-style":"default","site-sidebar-style":"default","ast-global-header-display":"","ast-banner-title-visibility":"","ast-main-header-display":"","ast-hfb-above-header-display":"","ast-hfb-below-header-display":"","ast-hfb-mobile-header-display":"","site-post-title":"","ast-breadcrumbs-content":"","ast-featured-img":"","footer-sml-layout":"","theme-transparent-header-meta":"","adv-header-id-meta":"","stick-header-meta":"","header-above-stick-meta":"","header-main-stick-meta":"","header-below-stick-meta":"","astra-migrate-meta-layouts":"default","ast-page-background-enabled":"default","ast-page-background-meta":{"desktop":{"background-color":"var(--ast-global-color-4)","background-image":"","background-repeat":"repeat","background-position":"center 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