{"id":139543,"date":"2026-01-12T17:46:32","date_gmt":"2026-01-12T17:46:32","guid":{"rendered":"https:\/\/www.deberes.net\/tesis\/sin-categoria\/cinetica-celular-y-su-regulacion-en-cancer-de-mama\/"},"modified":"2026-01-12T17:46:32","modified_gmt":"2026-01-12T17:46:32","slug":"cinetica-celular-y-su-regulacion-en-cancer-de-mama","status":"publish","type":"post","link":"https:\/\/www.deberes.net\/tesis\/ciencias-medicas\/cinetica-celular-y-su-regulacion-en-cancer-de-mama\/","title":{"rendered":"\u00abcinetica celular y su regulacion en cancer de mama\u00bb."},"content":{"rendered":"<h2>Tesis doctoral de <strong> Pablo Gonzalvo Rodriguez <\/strong><\/h2>\n<p>Fundamento teorico: el fenomeno de la oroliferacion en la neoplasia esta gobernado por sistemas cualitativa y cuantitativamente distintos de los mecanismos de regulacion en condiciones fisiologicas. Nuestro proposito es analizar los cambios cineticos en la neoplasia y para ello, hemos elegido como modelo el cancer de mama, dada su elevada frecuencia, accesibilidad quirurgica y los conocimientos recogidos en los ultimos tiempos sobre su control proliferativo.  modelo teorico: la velocidad del ciclo en una neoplasia es un factor determinante de la proliferacion. Segun se desprende de nuestra hipotesis, este parametro, puede ser estimado en material tisular y con este fin hemos elaborado un indice (indice de duracion de ciclo) expresado como el cociente entre el indice de mitosis y el indice proliferativo ki 67.  resultados: el analisis comparativo y de relacion del indice de duracion del ciclo, con otros parametros cineticos y pronosticos ha desmostrado, no solo una evolucion paralela al comportamiento proliferativo sino tambien una asociacion estrecha con la agresividad de la neoplasia.<\/p>\n<p>&nbsp;<\/p>\n<h3>Datos acad\u00e9micos de la tesis doctoral \u00ab<strong>\u00abcinetica celular y su regulacion en cancer de mama\u00bb.<\/strong>\u00ab<\/h3>\n<ul>\n<li><strong>T\u00edtulo de la tesis:<\/strong>\u00a0 \u00abcinetica celular y su regulacion en cancer de mama\u00bb. <\/li>\n<li><strong>Autor:<\/strong>\u00a0 Pablo Gonzalvo Rodriguez <\/li>\n<li><strong>Universidad:<\/strong>\u00a0 Oviedo<\/li>\n<li><strong>Fecha de lectura de la tesis:<\/strong>\u00a0 01\/01\/1992<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n<h3>Direcci\u00f3n y tribunal<\/h3>\n<ul>\n<li><strong>Director de la tesis<\/strong>\n<ul>\n<li>Andres Sampedro Nu\u00f1o<\/li>\n<\/ul>\n<\/li>\n<li><strong>Tribunal<\/strong>\n<ul>\n<li>Presidente del tribunal: Juan  Sebastian Lopez Arranz <\/li>\n<li>Horacio Oliva Aldamiz (vocal)<\/li>\n<li>Cesar Aguirre Viani (vocal)<\/li>\n<li>Carlos Lopez Otin (vocal)<\/li>\n<\/ul>\n<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n","protected":false},"excerpt":{"rendered":"<p>Tesis doctoral de Pablo Gonzalvo Rodriguez Fundamento teorico: el fenomeno de la oroliferacion en la neoplasia esta gobernado por sistemas [&hellip;]<\/p>\n","protected":false},"author":1,"featured_media":0,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"site-sidebar-layout":"default","site-content-layout":"","ast-site-content-layout":"","site-content-style":"default","site-sidebar-style":"default","ast-global-header-display":"","ast-banner-title-visibility":"","ast-main-header-display":"","ast-hfb-above-header-display":"","ast-hfb-below-header-display":"","ast-hfb-mobile-header-display":"","site-post-title":"","ast-breadcrumbs-content":"","ast-featured-img":"","footer-sml-layout":"","theme-transparent-header-meta":"","adv-header-id-meta":"","stick-header-meta":"","header-above-stick-meta":"","header-main-stick-meta":"","header-below-stick-meta":"","astra-migrate-meta-layouts":"default","ast-page-background-enabled":"default","ast-page-background-meta":{"desktop":{"background-color":"var(--ast-global-color-4)","background-image":"","background-repeat":"repeat","background-position":"center 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