{"id":143476,"date":"1993-01-01T00:00:00","date_gmt":"1993-01-01T00:00:00","guid":{"rendered":"https:\/\/www.deberes.net\/tesis\/sin-categoria\/glicosilacion-no-enzimatica-en-la-diabetes-estudios-in-vitro-e-in-vivo\/"},"modified":"1993-01-01T00:00:00","modified_gmt":"1993-01-01T00:00:00","slug":"glicosilacion-no-enzimatica-en-la-diabetes-estudios-in-vitro-e-in-vivo","status":"publish","type":"post","link":"https:\/\/www.deberes.net\/tesis\/ciencias-medicas\/glicosilacion-no-enzimatica-en-la-diabetes-estudios-in-vitro-e-in-vivo\/","title":{"rendered":"Glicosilacion no enzimatica en la diabetes. estudios \u00abin vitro\u00bb e \u00abin vivo\u00bb."},"content":{"rendered":"<h2>Tesis doctoral de <strong> Isabel Alonso Troncoso <\/strong><\/h2>\n<p>Los productos terminales de la glicosilacion no enzimatica (ages) se han implicado en la patogenesis de las complicaciones cronicas de la diabetes mellitus. \u00abIn vitro\u00bb la cantidad de ages formados esta en asociacion con el tiempo de exposicion a un medio glucosado de los tejidos y es inhibido totalmente por la adicion de aminoguanidina al medio. La aminoguanidina altera el aspecto macroscopico del tejido. Asi mismo \u00abin vivo\u00bb la cantidad de ages formados esta en asociacion con la edad.  en el estado diabetico, a igualdad de la edad, aumenta la cantidad de ages presente en los tejidos. El trasplante precoz de islotes revierte el estado catabolico de la rata diabetica e inhibe totalmente la formacion de ages.  nuestros datos sugieren que un tratamiento precoz con amg desde el diagnostico de la diabetes mellitus hasta el momento de practicar un trasplante precoz de tejido insular pudiera ser el tratamiento secuencial ideal. \u00abIn vivo\u00bb la fluorescencia podria no ser un indice especifico de ages.<\/p>\n<p>&nbsp;<\/p>\n<h3>Datos acad\u00e9micos de la tesis doctoral \u00ab<strong>Glicosilacion no enzimatica en la diabetes. estudios \u00abin vitro\u00bb e \u00abin vivo\u00bb.<\/strong>\u00ab<\/h3>\n<ul>\n<li><strong>T\u00edtulo de la tesis:<\/strong>\u00a0 Glicosilacion no enzimatica en la diabetes. estudios \u00abin vitro\u00bb e \u00abin vivo\u00bb. <\/li>\n<li><strong>Autor:<\/strong>\u00a0 Isabel Alonso Troncoso <\/li>\n<li><strong>Universidad:<\/strong>\u00a0 Oviedo<\/li>\n<li><strong>Fecha de lectura de la tesis:<\/strong>\u00a0 01\/01\/1993<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n<h3>Direcci\u00f3n y tribunal<\/h3>\n<ul>\n<li><strong>Director de la tesis<\/strong>\n<ul>\n<li>Manuel Mart\u00ednez Esteban<\/li>\n<\/ul>\n<\/li>\n<li><strong>Tribunal<\/strong>\n<ul>\n<li>Presidente del tribunal: Enrique Martinez Rodriguez <\/li>\n<li>Norberto Hernandez Siberio (vocal)<\/li>\n<li>Enrique Bl\u00e1zquez Fern\u00e1ndez (vocal)<\/li>\n<li>Eva Vidal V\u00e1zquez (vocal)<\/li>\n<\/ul>\n<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n","protected":false},"excerpt":{"rendered":"<p>Tesis doctoral de Isabel Alonso Troncoso Los productos terminales de la glicosilacion no enzimatica (ages) se han implicado en la [&hellip;]<\/p>\n","protected":false},"author":1,"featured_media":0,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"site-sidebar-layout":"default","site-content-layout":"","ast-site-content-layout":"","site-content-style":"default","site-sidebar-style":"default","ast-global-header-display":"","ast-banner-title-visibility":"","ast-main-header-display":"","ast-hfb-above-header-display":"","ast-hfb-below-header-display":"","ast-hfb-mobile-header-display":"","site-post-title":"","ast-breadcrumbs-content":"","ast-featured-img":"","footer-sml-layout":"","theme-transparent-header-meta":"","adv-header-id-meta":"","stick-header-meta":"","header-above-stick-meta":"","header-main-stick-meta":"","header-below-stick-meta":"","astra-migrate-meta-layouts":"default","ast-page-background-enabled":"default","ast-page-background-meta":{"desktop":{"background-color":"var(--ast-global-color-4)","background-image":"","background-repeat":"repeat","background-position":"center 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