{"id":145059,"date":"1993-01-01T00:00:00","date_gmt":"1993-01-01T00:00:00","guid":{"rendered":"https:\/\/www.deberes.net\/tesis\/sin-categoria\/hipoxia-e-hiperoxia-en-la-etiopatogenia-de-la-arteriosclerosis\/"},"modified":"1993-01-01T00:00:00","modified_gmt":"1993-01-01T00:00:00","slug":"hipoxia-e-hiperoxia-en-la-etiopatogenia-de-la-arteriosclerosis","status":"publish","type":"post","link":"https:\/\/www.deberes.net\/tesis\/ciencias-medicas\/hipoxia-e-hiperoxia-en-la-etiopatogenia-de-la-arteriosclerosis\/","title":{"rendered":"Hipoxia e hiperoxia en la etiopatogenia de la arteriosclerosis."},"content":{"rendered":"<h2>Tesis doctoral de <strong>  Rodriguez Cristobal Juan  Jose <\/strong><\/h2>\n<p>Objetivos: confirmar el papel de la hipoxia en la etiopatogenia de la arteriosclerosis y el valor profilactico o nocivo de la oxigenacion. Metodos:  experimentales con ratas comparando un modelo bien caracterizado, inducido por la administracion de vitamina d mas una dieta hiperlipidica, con situaciones especificas de anoxia aguda, anoxia cronica e hipoxia cronica, en camaras especialmente dise\u00f1adas y construidas para este fin, en ratas con hiperlipemia o sometidas al modelo de comparacion. Se evaluaron lesiones morfologicas, parametros lipidicos, del sistema fibrinolitico y agregacion plaquetaria.  resultados y conclusiones: la hipoxia aunque no se ha comportado como un cofactor aterogeno, puede favorecer el desarrollo de procesos tromboticos al disminuir la actividad fibrinolitica. La hiperoxia aumenta la resistencia a la induccion de lesiones arterioscleroticas en la rata, lo cual le confiere un cierto papel protector al mejorar las condiciones de los animales en su perfil lipidico y disminuir su potencial trombotico.<\/p>\n<p>&nbsp;<\/p>\n<h3>Datos acad\u00e9micos de la tesis doctoral \u00ab<strong>Hipoxia e hiperoxia en la etiopatogenia de la arteriosclerosis.<\/strong>\u00ab<\/h3>\n<ul>\n<li><strong>T\u00edtulo de la tesis:<\/strong>\u00a0 Hipoxia e hiperoxia en la etiopatogenia de la arteriosclerosis. <\/li>\n<li><strong>Autor:<\/strong>\u00a0  Rodriguez Cristobal Juan  Jose <\/li>\n<li><strong>Universidad:<\/strong>\u00a0 Aut\u00f3noma de barcelona<\/li>\n<li><strong>Fecha de lectura de la tesis:<\/strong>\u00a0 01\/01\/1993<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n<h3>Direcci\u00f3n y tribunal<\/h3>\n<ul>\n<li><strong>Director de la tesis<\/strong>\n<ul>\n<li>villaverde Grote Alonso<\/li>\n<\/ul>\n<\/li>\n<li><strong>Tribunal<\/strong>\n<ul>\n<li>Presidente del tribunal: Juan Rubies Prat <\/li>\n<li>Gin\u00e9s Escolar Albadalejo (vocal)<\/li>\n<li>Lina Badimon Maestro (vocal)<\/li>\n<li>Celestino Barastegui Almagro (vocal)<\/li>\n<\/ul>\n<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n","protected":false},"excerpt":{"rendered":"<p>Tesis doctoral de Rodriguez Cristobal Juan Jose Objetivos: confirmar el papel de la hipoxia en la etiopatogenia de la arteriosclerosis [&hellip;]<\/p>\n","protected":false},"author":1,"featured_media":0,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"site-sidebar-layout":"default","site-content-layout":"","ast-site-content-layout":"","site-content-style":"default","site-sidebar-style":"default","ast-global-header-display":"","ast-banner-title-visibility":"","ast-main-header-display":"","ast-hfb-above-header-display":"","ast-hfb-below-header-display":"","ast-hfb-mobile-header-display":"","site-post-title":"","ast-breadcrumbs-content":"","ast-featured-img":"","footer-sml-layout":"","theme-transparent-header-meta":"","adv-header-id-meta":"","stick-header-meta":"","header-above-stick-meta":"","header-main-stick-meta":"","header-below-stick-meta":"","astra-migrate-meta-layouts":"default","ast-page-background-enabled":"default","ast-page-background-meta":{"desktop":{"background-color":"var(--ast-global-color-4)","background-image":"","background-repeat":"repeat","background-position":"center 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