{"id":2924,"date":"1994-01-01T00:00:00","date_gmt":"1994-01-01T00:00:00","guid":{"rendered":"https:\/\/www.deberes.net\/tesis\/1994\/01\/01\/estudio-farmacologico-de-la-accion-de-leucotrienos-sobre-vasos-pulmonares-humanos-in-vitro\/"},"modified":"1994-01-01T00:00:00","modified_gmt":"1994-01-01T00:00:00","slug":"estudio-farmacologico-de-la-accion-de-leucotrienos-sobre-vasos-pulmonares-humanos-in-vitro","status":"publish","type":"post","link":"https:\/\/www.deberes.net\/tesis\/ciencias-medicas\/estudio-farmacologico-de-la-accion-de-leucotrienos-sobre-vasos-pulmonares-humanos-in-vitro\/","title":{"rendered":"Estudio farmacologico de la accion de leucotrienos sobre vasos pulmonares humanos in vitro"},"content":{"rendered":"<h2>Tesis doctoral de <strong> Amparo Seller Losada <\/strong><\/h2>\n<p>Los peptidoleucotrienos c4, d4, y e4 producen relajaciones endotelio-dependientes en arteria y vena pulmonar aisladas. Dichas relajaciones estan mediadas por la activacion de un receptor no pl1 situado en la celula endotelial. En arteria pulmonar la relajacion se produce por liberacion de oxido nitrico y prostaciclina, mientras en la vena pulmonar se libera unicamente oxido nitrico.  el leucotrieno d4 contrae la vena pulmonar humana aislada por activacion de un receptor pl2 situado en la celula muscular y por activacion de un receptor pl1 situado en la celula endotelial, a traves de la liberacion de un factor contracturante derivado del endotelio.<\/p>\n<p>&nbsp;<\/p>\n<h3>Datos acad\u00e9micos de la tesis doctoral \u00ab<strong>Estudio farmacologico de la accion de leucotrienos sobre vasos pulmonares humanos in vitro<\/strong>\u00ab<\/h3>\n<ul>\n<li><strong>T\u00edtulo de la tesis:<\/strong>\u00a0 Estudio farmacologico de la accion de leucotrienos sobre vasos pulmonares humanos in vitro <\/li>\n<li><strong>Autor:<\/strong>\u00a0 Amparo Seller Losada <\/li>\n<li><strong>Universidad:<\/strong>\u00a0 Universitat de val\u00e9ncia (estudi general)<\/li>\n<li><strong>Fecha de lectura de la tesis:<\/strong>\u00a0 01\/01\/1994<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n<h3>Direcci\u00f3n y tribunal<\/h3>\n<ul>\n<li><strong>Director de la tesis<\/strong>\n<ul>\n<li> Ortiz Belda Jos\u00e9 Luis<\/li>\n<\/ul>\n<\/li>\n<li><strong>Tribunal<\/strong>\n<ul>\n<li>Presidente del tribunal: Francisco Gomar Sancho <\/li>\n<li> Sanchez De La Cuesta Y Alarcon Felipe (vocal)<\/li>\n<li>Salvador Lluch Lopez (vocal)<\/li>\n<li>Luis San Rom\u00e1n Del Barrio (vocal)<\/li>\n<\/ul>\n<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n","protected":false},"excerpt":{"rendered":"<p>Tesis doctoral de Amparo Seller Losada Los peptidoleucotrienos c4, d4, y e4 producen relajaciones endotelio-dependientes en arteria y vena pulmonar [&hellip;]<\/p>\n","protected":false},"author":1,"featured_media":0,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"site-sidebar-layout":"default","site-content-layout":"","ast-site-content-layout":"","site-content-style":"default","site-sidebar-style":"default","ast-global-header-display":"","ast-banner-title-visibility":"","ast-main-header-display":"","ast-hfb-above-header-display":"","ast-hfb-below-header-display":"","ast-hfb-mobile-header-display":"","site-post-title":"","ast-breadcrumbs-content":"","ast-featured-img":"","footer-sml-layout":"","theme-transparent-header-meta":"","adv-header-id-meta":"","stick-header-meta":"","header-above-stick-meta":"","header-main-stick-meta":"","header-below-stick-meta":"","astra-migrate-meta-layouts":"default","ast-page-background-enabled":"default","ast-page-background-meta":{"desktop":{"background-color":"var(--ast-global-color-4)","background-image":"","background-repeat":"repeat","background-position":"center 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