{"id":41599,"date":"1999-01-01T00:00:00","date_gmt":"1999-01-01T00:00:00","guid":{"rendered":"https:\/\/www.deberes.net\/tesis\/sin-categoria\/infeccion-por-virus-g-en-ninos-con-hepatitis-cronica-viral\/"},"modified":"1999-01-01T00:00:00","modified_gmt":"1999-01-01T00:00:00","slug":"infeccion-por-virus-g-en-ninos-con-hepatitis-cronica-viral","status":"publish","type":"post","link":"https:\/\/www.deberes.net\/tesis\/ciencias-medicas\/infeccion-por-virus-g-en-ninos-con-hepatitis-cronica-viral\/","title":{"rendered":"Infeccion por virus g en ni\u00f1os con hepatitis cronica viral."},"content":{"rendered":"<h2>Tesis doctoral de <strong> Antonio Millan Jimenez <\/strong><\/h2>\n<p>Antecedentes: la existencia de un 10-20% de hapatitis de etiologi\u00eda no aclarada y la evidencia de que otros virus desconocidos hasta ahora podr\u00edan estar implicados en su etiolog\u00eda han impulsado la investigaci\u00f3n virol\u00f3gica en este campo. Recientemente se ha identificado un nuevo virus denominado virus g de la hepatitis (vgh) o virus gb tipo c(gbv-c). Se trata de un virus arn de cadena positiva perteneciente a la familia flaviviridae. Posteriormente ha sido aislado en el suero de pacientes con hepatitis no a-e, politransfundidos, consumidores de drogas por v\u00eda parenteral, transplantados y en pacientes con hepatitis b o c. Es, por tanto, evidente su transmisi\u00f3n parenteral. Sin embargo no lo es su capacidad para producir necrosis hep\u00e1tica o empeorar la evoluci\u00f3n de aquellos pacientes infectados por virus b(vbh) o c(vch).  objetivo: la finalidad del presente estudio es conocer la preValencia del vgh\/gbv-c en suero y tejido hep\u00e1tico, genotipo, v\u00edas de transmisi\u00f3n, respuesta al tratamiento con interfer\u00f3n y si influye en el grado de lesi\u00f3n hep\u00e1tica en ni\u00f1os con hepatitis cr\u00f3nica por vbh o vch.  m\u00e9todos: se incluyeron 58 ni\u00f1os con hepatitis cr\u00f3nica histol\u00f3gicamente probada, 33 por vbh y 25 por vch. La presencia del vgh\/gbv-c se estudi\u00f3, tanto en suero como en tejido hep\u00e1tico, mediante la t\u00e9cnica de reacci\u00f3n en cadena de la polimerasa (pcr). Se compararon los niveles de alanina aminotransferasa (alat), \u00edndice de actividad histol\u00f3gica de la lesi\u00f3n hep\u00e1tica y estadio de la fibrosis en aquellos coinfectados por el vgh\/gbv-c frente a los que no lo estaban. En los infectados por el vgh\/gbv-c se determin\u00f3 su presencia en los familiares de primer grado y se realiz\u00f3 un estudio filogen\u00e9tico y de homolog\u00eda de la secuencia del rna del vgh\/gbv-c entre los miembros de las familias. Asimisno, se estudiaron muestras de sueros congelados, obtenidos antes y tras recibir tratamiento con interfer\u00f3n, perteneciente a tres ni\u00f1o<\/p>\n<p>&nbsp;<\/p>\n<h3>Datos acad\u00e9micos de la tesis doctoral \u00ab<strong>Infeccion por virus g en ni\u00f1os con hepatitis cronica viral.<\/strong>\u00ab<\/h3>\n<ul>\n<li><strong>T\u00edtulo de la tesis:<\/strong>\u00a0 Infeccion por virus g en ni\u00f1os con hepatitis cronica viral. <\/li>\n<li><strong>Autor:<\/strong>\u00a0 Antonio Millan Jimenez <\/li>\n<li><strong>Universidad:<\/strong>\u00a0 Aut\u00f3noma de Madrid<\/li>\n<li><strong>Fecha de lectura de la tesis:<\/strong>\u00a0 01\/01\/1999<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n<h3>Direcci\u00f3n y tribunal<\/h3>\n<ul>\n<li><strong>Director de la tesis<\/strong>\n<ul>\n<li>Mercedes Ruiz Moreno<\/li>\n<\/ul>\n<\/li>\n<li><strong>Tribunal<\/strong>\n<ul>\n<li>Presidente del tribunal: manuel Hern\u00e1ndez rodr\u00edguez <\/li>\n<li>Javier Bartolome nebrada (vocal)<\/li>\n<li>modesto Lopez-linares prado (vocal)<\/li>\n<li>Jaime Gosalvez berenguer (vocal)<\/li>\n<\/ul>\n<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n","protected":false},"excerpt":{"rendered":"<p>Tesis doctoral de Antonio Millan Jimenez Antecedentes: la existencia de un 10-20% de hapatitis de etiologi\u00eda no aclarada y la 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