{"id":45805,"date":"2019-07-21T10:59:41","date_gmt":"2019-07-21T10:59:41","guid":{"rendered":"https:\/\/www.deberes.net\/tesis\/sin-categoria\/dopamina-beta-hidroxilasa-y-neurotrasmisores-en-hipertension-arterial-esencial\/"},"modified":"2019-07-21T10:59:41","modified_gmt":"2019-07-21T10:59:41","slug":"dopamina-beta-hidroxilasa-y-neurotrasmisores-en-hipertension-arterial-esencial","status":"publish","type":"post","link":"https:\/\/www.deberes.net\/tesis\/ciencias-agrarias\/dopamina-beta-hidroxilasa-y-neurotrasmisores-en-hipertension-arterial-esencial\/","title":{"rendered":"Dopamina beta hidroxilasa y neurotrasmisores en hipertension arterial esencial."},"content":{"rendered":"<h2>Tesis doctoral de <strong>  Prieto Martinez Juan  Manuel <\/strong><\/h2>\n<p>Siendo el incremento de las resistencias vasculares perifericas mecanismo basico en la etiopatogenia de la h.A. Y no conociendose bien el mecanismo del incremento de resistencias vasculares un gran cuerpo de doctrina mantiene la tesis del papel de mayores influencias adrenergicas sobre los receptores apropiados a nivel de las celulas musculares lisas de las arteriolas. Dado que las influencias dopaminergicas tienen un papel prevalente en la vasoconstriccionarteriolar y que el enzima dbh cataliza el paso desde dopamina a noradrenalina  hemos estudiado los niveles sericos de dbh en un conjunto de 256 pacientes de los que 104 tenian hipertension arterial y el resto eran normotensos actuando como controles con niveles normales de la enzima los restantes sujetos. La bondad del metodo quedo probada por el alto nivel de la enzima en un caso de feocromocitoma con h.A.<\/p>\n<p>&nbsp;<\/p>\n<h3>Datos acad\u00e9micos de la tesis doctoral \u00ab<strong>Dopamina beta hidroxilasa y neurotrasmisores en hipertension arterial esencial.<\/strong>\u00ab<\/h3>\n<ul>\n<li><strong>T\u00edtulo de la tesis:<\/strong>\u00a0 Dopamina beta hidroxilasa y neurotrasmisores en hipertension arterial esencial. <\/li>\n<li><strong>Autor:<\/strong>\u00a0  Prieto Martinez Juan  Manuel <\/li>\n<li><strong>Universidad:<\/strong>\u00a0 Sevilla<\/li>\n<li><strong>Fecha de lectura de la tesis:<\/strong>\u00a0 01\/01\/1979<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n<h3>Direcci\u00f3n y tribunal<\/h3>\n<ul>\n<li><strong>Director de la tesis<\/strong>\n<ul>\n<li>Miguel Garrido Peralta<\/li>\n<\/ul>\n<\/li>\n<li><strong>Tribunal<\/strong>\n<ul>\n<li>Presidente del tribunal: Raimundo Goberna Ortiz <\/li>\n<li>Miguel Garrido Peralta (vocal)<\/li>\n<li>Antonio Aznar Reig (vocal)<\/li>\n<li>Enrique Romero Velasco (vocal)<\/li>\n<\/ul>\n<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n","protected":false},"excerpt":{"rendered":"<p>Tesis doctoral de Prieto Martinez Juan Manuel Siendo el incremento de las resistencias vasculares perifericas mecanismo basico en la etiopatogenia [&hellip;]<\/p>\n","protected":false},"author":1,"featured_media":0,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"site-sidebar-layout":"default","site-content-layout":"","ast-site-content-layout":"","site-content-style":"default","site-sidebar-style":"default","ast-global-header-display":"","ast-banner-title-visibility":"","ast-main-header-display":"","ast-hfb-above-header-display":"","ast-hfb-below-header-display":"","ast-hfb-mobile-header-display":"","site-post-title":"","ast-breadcrumbs-content":"","ast-featured-img":"","footer-sml-layout":"","theme-transparent-header-meta":"","adv-header-id-meta":"","stick-header-meta":"","header-above-stick-meta":"","header-main-stick-meta":"","header-below-stick-meta":"","astra-migrate-meta-layouts":"default","ast-page-background-enabled":"default","ast-page-background-meta":{"desktop":{"background-color":"var(--ast-global-color-4)","background-image":"","background-repeat":"repeat","background-position":"center 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