{"id":47359,"date":"2020-04-08T14:46:03","date_gmt":"2020-04-08T14:46:03","guid":{"rendered":"https:\/\/www.deberes.net\/tesis\/sin-categoria\/sintesis-y-secrecion-de-hormona-de-crecimiento-en-la-diabetes-experimental\/"},"modified":"2020-04-08T14:46:03","modified_gmt":"2020-04-08T14:46:03","slug":"sintesis-y-secrecion-de-hormona-de-crecimiento-en-la-diabetes-experimental","status":"publish","type":"post","link":"https:\/\/www.deberes.net\/tesis\/quimica\/sintesis-y-secrecion-de-hormona-de-crecimiento-en-la-diabetes-experimental\/","title":{"rendered":"Sintesis y secrecion de hormona de crecimiento en la diabetes experimental."},"content":{"rendered":"<h2>Tesis doctoral de <strong> Concepcion Gonzalez Marquez <\/strong><\/h2>\n<p>En ratas en fase de crecimiento  la induccion de la diabetes origina un retraso en el crecimiento acompa\u00f1ado de un descenso en el contenido hipofisario y en la sintesis de novo de hormona de crecimiento (gh). Los efectos observados no son inespecificos de la droga usada (streptozotocin) como agente diabetogeno ya que son reversibles por administracion de insulina. La concentracion circulante de gh en condiciones basales no difiere entre controles y diabeticos.Por el contrario  en situaciones de stress (anestesia con eter o con pentobarbital)  los animales diabeticos muestran una hipersecrecion hipofisaria de gh  efecto que no se observa en condiciones basales. Tanto el contenido hipofisario como los niveles circulantes de gh se han medido por radioinmunoensayo (ria).<\/p>\n<p>&nbsp;<\/p>\n<h3>Datos acad\u00e9micos de la tesis doctoral \u00ab<strong>Sintesis y secrecion de hormona de crecimiento en la diabetes experimental.<\/strong>\u00ab<\/h3>\n<ul>\n<li><strong>T\u00edtulo de la tesis:<\/strong>\u00a0 Sintesis y secrecion de hormona de crecimiento en la diabetes experimental. <\/li>\n<li><strong>Autor:<\/strong>\u00a0 Concepcion Gonzalez Marquez <\/li>\n<li><strong>Universidad:<\/strong>\u00a0 Aut\u00f3noma de Madrid<\/li>\n<li><strong>Fecha de lectura de la tesis:<\/strong>\u00a0 01\/01\/1980<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n<h3>Direcci\u00f3n y tribunal<\/h3>\n<ul>\n<li><strong>Director de la tesis<\/strong>\n<ul>\n<li>Trinidad Tolin Buzo<\/li>\n<\/ul>\n<\/li>\n<li><strong>Tribunal<\/strong>\n<ul>\n<li>Presidente del tribunal: Arsenio Fraile Ovejero <\/li>\n<li>Emilio Herrera Castillon (vocal)<\/li>\n<li>Amadeo Sa\u00f1udo Palazuelos (vocal)<\/li>\n<li> Escobar Del Rey  Francisco (vocal)<\/li>\n<\/ul>\n<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n","protected":false},"excerpt":{"rendered":"<p>Tesis doctoral de Concepcion Gonzalez Marquez En ratas en fase de crecimiento la induccion de la diabetes origina un retraso [&hellip;]<\/p>\n","protected":false},"author":1,"featured_media":0,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"site-sidebar-layout":"default","site-content-layout":"","ast-site-content-layout":"","site-content-style":"default","site-sidebar-style":"default","ast-global-header-display":"","ast-banner-title-visibility":"","ast-main-header-display":"","ast-hfb-above-header-display":"","ast-hfb-below-header-display":"","ast-hfb-mobile-header-display":"","site-post-title":"","ast-breadcrumbs-content":"","ast-featured-img":"","footer-sml-layout":"","theme-transparent-header-meta":"","adv-header-id-meta":"","stick-header-meta":"","header-above-stick-meta":"","header-main-stick-meta":"","header-below-stick-meta":"","astra-migrate-meta-layouts":"default","ast-page-background-enabled":"default","ast-page-background-meta":{"desktop":{"background-color":"var(--ast-global-color-4)","background-image":"","background-repeat":"repeat","background-position":"center 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