{"id":49113,"date":"2021-01-26T01:51:56","date_gmt":"2021-01-26T01:51:56","guid":{"rendered":"https:\/\/www.deberes.net\/tesis\/sin-categoria\/energia-cardiocirculatoria-mecanismos-de-compensacion-de-los-pacientes-cardiopatas-y-posibilidades-terapeuticas\/"},"modified":"2021-01-26T01:51:56","modified_gmt":"2021-01-26T01:51:56","slug":"energia-cardiocirculatoria-mecanismos-de-compensacion-de-los-pacientes-cardiopatas-y-posibilidades-terapeuticas","status":"publish","type":"post","link":"https:\/\/www.deberes.net\/tesis\/ciencias-medicas\/energia-cardiocirculatoria-mecanismos-de-compensacion-de-los-pacientes-cardiopatas-y-posibilidades-terapeuticas\/","title":{"rendered":"Energia cardiocirculatoria. mecanismos de compensacion de los pacientes cardiopatas y posibilidades terapeuticas."},"content":{"rendered":"<h2>Tesis doctoral de <strong>  San Vicente Urondo Ludmila <\/strong><\/h2>\n<p>Este trabajo se basa en los datos clinicos y hemodinamicos cardiacos de 726 sujetos. En el se ha medido la energia potencial del aparato cardiocirculatorio (w x m-2) de 707 individuos normales y cardiopatas    y se ha establecido una relacion  entre el nivel de energia y el indice de volumen sistolico comprobandose que no existe una relacion lineal entre ambos. Ello es debido a la puesta en marcha de mecanismos de compensacion simpatica con aumento de las resistencias vasculares perifericas y de la frecuencia cardiaca. Se ha cuantificado la magnitud de la respuesta compensadora por accion de la regulacion neurovegetativa y por accion central (hipertrofia y dilatacion cardiaca) de lo que pueden extraerse algunas conclusiones terapeuticas. Se ha comprobado que el incremento de las resistencias se produce de forma distinta en los pacientes cardiopatas y en los hipertensos.<\/p>\n<p>&nbsp;<\/p>\n<h3>Datos acad\u00e9micos de la tesis doctoral \u00ab<strong>Energia cardiocirculatoria. mecanismos de compensacion de los pacientes cardiopatas y posibilidades terapeuticas.<\/strong>\u00ab<\/h3>\n<ul>\n<li><strong>T\u00edtulo de la tesis:<\/strong>\u00a0 Energia cardiocirculatoria. mecanismos de compensacion de los pacientes cardiopatas y posibilidades terapeuticas. <\/li>\n<li><strong>Autor:<\/strong>\u00a0  San Vicente Urondo Ludmila <\/li>\n<li><strong>Universidad:<\/strong>\u00a0 Barcelona<\/li>\n<li><strong>Fecha de lectura de la tesis:<\/strong>\u00a0 01\/01\/1982<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n<h3>Direcci\u00f3n y tribunal<\/h3>\n<ul>\n<li><strong>Director de la tesis<\/strong>\n<ul>\n<li>Cirilo Rozman Borsmar<\/li>\n<\/ul>\n<\/li>\n<li><strong>Tribunal<\/strong>\n<ul>\n<li>Presidente del tribunal: Cirilo Rozman Borsmar <\/li>\n<li>Antonio Cardesa Garc\u00eda (vocal)<\/li>\n<li>Jos\u00e9 M Mascaro Ballester (vocal)<\/li>\n<li>Ricardo Castillo Cofi\u00f1o (vocal)<\/li>\n<\/ul>\n<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n","protected":false},"excerpt":{"rendered":"<p>Tesis doctoral de San Vicente Urondo Ludmila Este trabajo se basa en los datos clinicos y hemodinamicos cardiacos de 726 [&hellip;]<\/p>\n","protected":false},"author":1,"featured_media":0,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"site-sidebar-layout":"default","site-content-layout":"","ast-site-content-layout":"","site-content-style":"default","site-sidebar-style":"default","ast-global-header-display":"","ast-banner-title-visibility":"","ast-main-header-display":"","ast-hfb-above-header-display":"","ast-hfb-below-header-display":"","ast-hfb-mobile-header-display":"","site-post-title":"","ast-breadcrumbs-content":"","ast-featured-img":"","footer-sml-layout":"","theme-transparent-header-meta":"","adv-header-id-meta":"","stick-header-meta":"","header-above-stick-meta":"","header-main-stick-meta":"","header-below-stick-meta":"","astra-migrate-meta-layouts":"default","ast-page-background-enabled":"default","ast-page-background-meta":{"desktop":{"background-color":"var(--ast-global-color-4)","background-image":"","background-repeat":"repeat","background-position":"center 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