{"id":49563,"date":"2021-04-09T02:37:21","date_gmt":"2021-04-09T02:37:21","guid":{"rendered":"https:\/\/www.deberes.net\/tesis\/sin-categoria\/estudio-clinico-y-analitico-de-la-encefalopatia-pancreatica\/"},"modified":"2021-04-09T02:37:21","modified_gmt":"2021-04-09T02:37:21","slug":"estudio-clinico-y-analitico-de-la-encefalopatia-pancreatica","status":"publish","type":"post","link":"https:\/\/www.deberes.net\/tesis\/ciencias-medicas\/estudio-clinico-y-analitico-de-la-encefalopatia-pancreatica\/","title":{"rendered":"Estudio clinico y analitico de la encefalopatia pancreatica."},"content":{"rendered":"<h2>Tesis doctoral de <strong>  Moreno Moraga Francisco Javier <\/strong><\/h2>\n<p>Se ha realizado el estudio proyectivo de 22 casos de encefalopatia pancreatica comparando el curso clinico de la enfermedad de base  la pancreatitis aguda  con otros 43 enfermos sin alteraciones neuropsiquicas. El curso clinico fue el mismo y los criterios de valoracion de gravedad fueron similares. En la mitad de los casos la causa de la pancreatitis aguda fue una colecistopatia y 2\/3 partes fueron mujeres. Lo mas caracteristico del curso clinico es la agitacion y las alucinaciones visuales y auditivas. El eeg siempre estuvo alterado. En todos los casos y en los dos grupos controles se determino lipasa en sangre y lcr  presentando una clara elevacion en el grupo de emofalopatia (p(0 001)  sin relacion lineal con la lipasemia (p(0 5). La isolipasa pancreatica estuvo elevada en el suero de los enfermos de pancreatitis pero fue indetectable en el lcr de todos los grupos. La lipasa elevada en lcr es de utilidad diagnostica  interviene en la patogenia de la encefalopatia  pero no depende de la isolipasa pancreatica sino probablemente de su liberacion en el snc.<\/p>\n<p>&nbsp;<\/p>\n<h3>Datos acad\u00e9micos de la tesis doctoral \u00ab<strong>Estudio clinico y analitico de la encefalopatia pancreatica.<\/strong>\u00ab<\/h3>\n<ul>\n<li><strong>T\u00edtulo de la tesis:<\/strong>\u00a0 Estudio clinico y analitico de la encefalopatia pancreatica. <\/li>\n<li><strong>Autor:<\/strong>\u00a0  Moreno Moraga Francisco Javier <\/li>\n<li><strong>Universidad:<\/strong>\u00a0 Complutense de Madrid<\/li>\n<li><strong>Fecha de lectura de la tesis:<\/strong>\u00a0 01\/01\/1982<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n<h3>Direcci\u00f3n y tribunal<\/h3>\n<ul>\n<li><strong>Director de la tesis<\/strong>\n<ul>\n<li>Vicente Gilsanz Garcia<\/li>\n<\/ul>\n<\/li>\n<li><strong>Tribunal<\/strong>\n<ul>\n<li>Presidente del tribunal: Vicente Gilsanz Garcia <\/li>\n<li>Amador Schuller Perez (vocal)<\/li>\n<li>Jorge Tamarit Torres (vocal)<\/li>\n<li>Juliana Fari\u00f1as Gonzalez (vocal)<\/li>\n<\/ul>\n<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n","protected":false},"excerpt":{"rendered":"<p>Tesis doctoral de Moreno Moraga Francisco Javier Se ha realizado el estudio proyectivo de 22 casos de encefalopatia pancreatica comparando [&hellip;]<\/p>\n","protected":false},"author":1,"featured_media":0,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"site-sidebar-layout":"default","site-content-layout":"","ast-site-content-layout":"","site-content-style":"default","site-sidebar-style":"default","ast-global-header-display":"","ast-banner-title-visibility":"","ast-main-header-display":"","ast-hfb-above-header-display":"","ast-hfb-below-header-display":"","ast-hfb-mobile-header-display":"","site-post-title":"","ast-breadcrumbs-content":"","ast-featured-img":"","footer-sml-layout":"","theme-transparent-header-meta":"","adv-header-id-meta":"","stick-header-meta":"","header-above-stick-meta":"","header-main-stick-meta":"","header-below-stick-meta":"","astra-migrate-meta-layouts":"default","ast-page-background-enabled":"default","ast-page-background-meta":{"desktop":{"background-color":"var(--ast-global-color-4)","background-image":"","background-repeat":"repeat","background-position":"center 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