{"id":52751,"date":"2022-04-20T17:37:39","date_gmt":"2022-04-20T17:37:39","guid":{"rendered":"https:\/\/www.deberes.net\/tesis\/sin-categoria\/actividad-plasmatica-de-dopamina-beta-hidroxilasa-en-la-diabetes-mellitus-experimental\/"},"modified":"2022-04-20T17:37:39","modified_gmt":"2022-04-20T17:37:39","slug":"actividad-plasmatica-de-dopamina-beta-hidroxilasa-en-la-diabetes-mellitus-experimental","status":"publish","type":"post","link":"https:\/\/www.deberes.net\/tesis\/ciencias-medicas\/actividad-plasmatica-de-dopamina-beta-hidroxilasa-en-la-diabetes-mellitus-experimental\/","title":{"rendered":"Actividad plasmatica de dopamina-beta-hidroxilasa en la diabetes mellitus experimental."},"content":{"rendered":"<h2>Tesis doctoral de <strong>  Mu\u00f1oz Sanchez Jos\u00e9 Andres <\/strong><\/h2>\n<p>El incremento de la actividad plasmatica de la dopamina-b-hidroxilasa (a-dbh-p) ante el deficit de insulina en la diabetes experimental  se produce por un mecanismo secretor por hiperfuncion sinpato-adrenal y por una disminucion catabolica de la glicoproteina plasmatica dbh. Se encuentra una correlacion entre la actividad simpato-adrenal y la presentacion de cataratas. La accion bloqueante y competitiva de la hiperglucemia sobre el aclaramiento de la dbh plasmatica se ejerce por el azucar terminal manosa\/n- acetil glucosamina de la porcion carbohidratada de la glicoproteina y sobre el receptor catabolico manosa especifico. La a-dbh-p puede ser un indice pronostico y control del estado diabetico.<\/p>\n<p>&nbsp;<\/p>\n<h3>Datos acad\u00e9micos de la tesis doctoral \u00ab<strong>Actividad plasmatica de dopamina-beta-hidroxilasa en la diabetes mellitus experimental.<\/strong>\u00ab<\/h3>\n<ul>\n<li><strong>T\u00edtulo de la tesis:<\/strong>\u00a0 Actividad plasmatica de dopamina-beta-hidroxilasa en la diabetes mellitus experimental. <\/li>\n<li><strong>Autor:<\/strong>\u00a0  Mu\u00f1oz Sanchez Jos\u00e9 Andres <\/li>\n<li><strong>Universidad:<\/strong>\u00a0 Murcia<\/li>\n<li><strong>Fecha de lectura de la tesis:<\/strong>\u00a0 01\/01\/1984<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n<h3>Direcci\u00f3n y tribunal<\/h3>\n<ul>\n<li><strong>Director de la tesis<\/strong>\n<ul>\n<li>Tomas Quesada Perez<\/li>\n<\/ul>\n<\/li>\n<li><strong>Tribunal<\/strong>\n<ul>\n<li>Presidente del tribunal: Antonio Garcia Garcia <\/li>\n<li>Tomas Quesada Perez (vocal)<\/li>\n<li>Jos\u00e9 Enrique Campillo \u00e1lvarez (vocal)<\/li>\n<li>Mar\u00eda  Teresa Miras Portugal (vocal)<\/li>\n<\/ul>\n<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n","protected":false},"excerpt":{"rendered":"<p>Tesis doctoral de Mu\u00f1oz Sanchez Jos\u00e9 Andres El incremento de la actividad plasmatica de la dopamina-b-hidroxilasa (a-dbh-p) ante el deficit [&hellip;]<\/p>\n","protected":false},"author":1,"featured_media":0,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"site-sidebar-layout":"default","site-content-layout":"","ast-site-content-layout":"","site-content-style":"default","site-sidebar-style":"default","ast-global-header-display":"","ast-banner-title-visibility":"","ast-main-header-display":"","ast-hfb-above-header-display":"","ast-hfb-below-header-display":"","ast-hfb-mobile-header-display":"","site-post-title":"","ast-breadcrumbs-content":"","ast-featured-img":"","footer-sml-layout":"","theme-transparent-header-meta":"","adv-header-id-meta":"","stick-header-meta":"","header-above-stick-meta":"","header-main-stick-meta":"","header-below-stick-meta":"","astra-migrate-meta-layouts":"default","ast-page-background-enabled":"default","ast-page-background-meta":{"desktop":{"background-color":"var(--ast-global-color-4)","background-image":"","background-repeat":"repeat","background-position":"center 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