{"id":7404,"date":"1995-01-01T00:00:00","date_gmt":"1995-01-01T00:00:00","guid":{"rendered":"https:\/\/www.deberes.net\/tesis\/1995\/01\/01\/regulacion-de-la-secrecion-de-hormona-de-crecimiento-en-el-hipercortisolismo-endogeno-sindrome-de-cushing\/"},"modified":"1995-01-01T00:00:00","modified_gmt":"1995-01-01T00:00:00","slug":"regulacion-de-la-secrecion-de-hormona-de-crecimiento-en-el-hipercortisolismo-endogeno-sindrome-de-cushing","status":"publish","type":"post","link":"https:\/\/www.deberes.net\/tesis\/ciencias-medicas\/regulacion-de-la-secrecion-de-hormona-de-crecimiento-en-el-hipercortisolismo-endogeno-sindrome-de-cushing\/","title":{"rendered":"Regulacion de la secrecion de hormona de crecimiento en el hipercortisolismo endogeno (sindrome de cushing)."},"content":{"rendered":"<h2>Tesis doctoral de <strong> Alfonso Pumar Lopez <\/strong><\/h2>\n<p>La secrecion de hormona de crecimiento esta inhibida en el sindrome de cushing, sin embargo se desconocen los mecanismos exactos por los que el hipercortisolismo cronico determina dicha inhibicion. El objetivo de la presente tesis doctoral ha sido intentar aportar algun dato en el esclarecimiento de los posibles mecanismos implicados, y para ello se han llevado a cabo una serie de pruebas de manipulacion farmacologica en pacientes con sindrome de cushing.  de los resultados obtenidos, hemos concluido que el efecto del exceso cronico de los glucocorticoides sobre la inhibicion de la secrecion de hormona de crecimiento, esta mediado por una severa alteracion hipotalamica que comporta una disminucion de la secrecion de ghrh y probablemente un aumento de la de somatostatina, y por efecto directo a nivel hipofisario sobre las celulas somatotropas.<\/p>\n<p>&nbsp;<\/p>\n<h3>Datos acad\u00e9micos de la tesis doctoral \u00ab<strong>Regulacion de la secrecion de hormona de crecimiento en el hipercortisolismo endogeno (sindrome de cushing).<\/strong>\u00ab<\/h3>\n<ul>\n<li><strong>T\u00edtulo de la tesis:<\/strong>\u00a0 Regulacion de la secrecion de hormona de crecimiento en el hipercortisolismo endogeno (sindrome de cushing). <\/li>\n<li><strong>Autor:<\/strong>\u00a0 Alfonso Pumar Lopez <\/li>\n<li><strong>Universidad:<\/strong>\u00a0 Sevilla<\/li>\n<li><strong>Fecha de lectura de la tesis:<\/strong>\u00a0 01\/01\/1995<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n<h3>Direcci\u00f3n y tribunal<\/h3>\n<ul>\n<li><strong>Director de la tesis<\/strong>\n<ul>\n<li>Alfonso Leal Cerro<\/li>\n<\/ul>\n<\/li>\n<li><strong>Tribunal<\/strong>\n<ul>\n<li>Presidente del tribunal: Carlos Dieguez Gonzalez <\/li>\n<li>Luis Jimenez Reina (vocal)<\/li>\n<li>Felipe Casanueva Freijo (vocal)<\/li>\n<li>Pedro Pablo Garcia Luna (vocal)<\/li>\n<\/ul>\n<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n","protected":false},"excerpt":{"rendered":"<p>Tesis doctoral de Alfonso Pumar Lopez La secrecion de hormona de crecimiento esta inhibida en el sindrome de cushing, sin [&hellip;]<\/p>\n","protected":false},"author":1,"featured_media":0,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"site-sidebar-layout":"default","site-content-layout":"","ast-site-content-layout":"","site-content-style":"default","site-sidebar-style":"default","ast-global-header-display":"","ast-banner-title-visibility":"","ast-main-header-display":"","ast-hfb-above-header-display":"","ast-hfb-below-header-display":"","ast-hfb-mobile-header-display":"","site-post-title":"","ast-breadcrumbs-content":"","ast-featured-img":"","footer-sml-layout":"","theme-transparent-header-meta":"","adv-header-id-meta":"","stick-header-meta":"","header-above-stick-meta":"","header-main-stick-meta":"","header-below-stick-meta":"","astra-migrate-meta-layouts":"default","ast-page-background-enabled":"default","ast-page-background-meta":{"desktop":{"background-color":"var(--ast-global-color-4)","background-image":"","background-repeat":"repeat","background-position":"center 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