{"id":83554,"date":"2018-03-10T00:07:43","date_gmt":"2018-03-10T00:07:43","guid":{"rendered":"https:\/\/www.deberes.net\/tesis\/sin-categoria\/estudio-de-la-lesion-celular-miocardica-en-la-cardiopata%c2%ada-hipertensiva\/"},"modified":"2018-03-10T00:07:43","modified_gmt":"2018-03-10T00:07:43","slug":"estudio-de-la-lesion-celular-miocardica-en-la-cardiopata%c2%ada-hipertensiva","status":"publish","type":"post","link":"https:\/\/www.deberes.net\/tesis\/ciencias-medicas\/estudio-de-la-lesion-celular-miocardica-en-la-cardiopata%c2%ada-hipertensiva\/","title":{"rendered":"Estudio de la lesion celular mioc\u00e1rdica en la cardiopat\u00edahipertensiva"},"content":{"rendered":"<h2>Tesis doctoral de <strong> Guillermo Jaime Pons  Llad\u00f3 <\/strong><\/h2>\n<p>Es conocido el papel epidemiol\u00f3gico de la hipertensi\u00f3n arterial como factor de riesgo en la enfermedad arteriosclerosa, por una parte, y en la insuficiencia cardiaca congestiva, en este caso mediante el desarrollo de la deonominada cardiopat\u00eda hipertensiva. Existe evidencia de que \u00e9sta \u00faltima se produce como fase evolutiva de un proceso que inicialmente es compensatorio, en forma de hipertrofia ventricular, derivando finalmente en una insuficiencia mioc\u00e1rdica, la base de la cual es el desarrollo de una lesi\u00f3n celular mioc\u00e1rdica progresiva que se ha relacionado con la propia hipertrofia, y cuya causa \u00faltima se atribuye a diversos factores, como en la deprivaci\u00f3n energ\u00e9tica o fen\u00f3menos de apoptosis, pero cuya presencia y curso evolutivo no han sido demostrados en el hombre.  con el objetivo de estudiar estos aspectos, se practicaron estudios isot\u00f3picos de captaci\u00f3n de anticuerpos antimiosina marcados con 111 indio, una tecnica con capacidad demostrada para evidenciar la presencia i el grado de lesi\u00f3n celular mioc\u00e1rdica, en una serie de pacientes hipertensos. se estudiaron 39 enfermos, distribuidos en 3 grupos:i) 16 pacientes asintom\u00e1ticos sin hipertrofia ventricular izquierda enel estudio ecocardiogr\u00e1fico; ii) 14 igualmente asintom\u00e1ticos, con hipertrofia ventricular;y iii) 9 pacientes con cuadro cl\u00ednico de cardiopat\u00eda hipertensiva evolucionada, en forma de insuficiencia cardiaca o dolor tor\u00e1cico anginoso, si bien con arterias coronarias angiogr\u00e1ficamente normales. En todos ellos se correlacion\u00f3 el indice coraz\u00f3n\/pulm\u00f3n de captaci\u00f3n de antimiosina(normal hasta 1,55) con variables cl\u00ednicas y ecocardiogr\u00e1ficas, incluyendo la masa ventricular izquierda.  los resultados permitieron observar captaci\u00f3n anormal de antimiosina en 4\/16 pacientes del grupo i(25%) en 2\/14 del grupo ii (15%) y en 9\/9 del grupo iii(100%). El \u00edndice coraz\u00f3n\/pulm\u00f3n en el grupo iii(1,77+- 0.16)era significativamente superior que en los grup<\/p>\n<p>&nbsp;<\/p>\n<h3>Datos acad\u00e9micos de la tesis doctoral \u00ab<strong>Estudio de la lesion celular mioc\u00e1rdica en la cardiopat\u00edahipertensiva<\/strong>\u00ab<\/h3>\n<ul>\n<li><strong>T\u00edtulo de la tesis:<\/strong>\u00a0 Estudio de la lesion celular mioc\u00e1rdica en la cardiopat\u00edahipertensiva <\/li>\n<li><strong>Autor:<\/strong>\u00a0 Guillermo Jaime Pons  Llad\u00f3 <\/li>\n<li><strong>Universidad:<\/strong>\u00a0 Aut\u00f3noma de barcelona<\/li>\n<li><strong>Fecha de lectura de la tesis:<\/strong>\u00a0 25\/02\/2000<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n<h3>Direcci\u00f3n y tribunal<\/h3>\n<ul>\n<li><strong>Director de la tesis<\/strong>\n<ul>\n<li>Manel Ballester Rodes<\/li>\n<\/ul>\n<\/li>\n<li><strong>Tribunal<\/strong>\n<ul>\n<li>Presidente del tribunal: Antonio Bayes de luna <\/li>\n<li>ignasi Carrio gaset (vocal)<\/li>\n<li>Francisco Javier Laso guzman (vocal)<\/li>\n<li> Pare bardera joan carles (vocal)<\/li>\n<\/ul>\n<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n","protected":false},"excerpt":{"rendered":"<p>Tesis doctoral de Guillermo Jaime Pons Llad\u00f3 Es conocido el papel epidemiol\u00f3gico de la hipertensi\u00f3n arterial como factor de riesgo [&hellip;]<\/p>\n","protected":false},"author":1,"featured_media":0,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"site-sidebar-layout":"default","site-content-layout":"","ast-site-content-layout":"","site-content-style":"default","site-sidebar-style":"default","ast-global-header-display":"","ast-banner-title-visibility":"","ast-main-header-display":"","ast-hfb-above-header-display":"","ast-hfb-below-header-display":"","ast-hfb-mobile-header-display":"","site-post-title":"","ast-breadcrumbs-content":"","ast-featured-img":"","footer-sml-layout":"","theme-transparent-header-meta":"","adv-header-id-meta":"","stick-header-meta":"","header-above-stick-meta":"","header-main-stick-meta":"","header-below-stick-meta":"","astra-migrate-meta-layouts":"default","ast-page-background-enabled":"default","ast-page-background-meta":{"desktop":{"background-color":"var(--ast-global-color-4)","background-image":"","background-repeat":"repeat","background-position":"center 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