{"id":8632,"date":"1995-01-01T00:00:00","date_gmt":"1995-01-01T00:00:00","guid":{"rendered":"https:\/\/www.deberes.net\/tesis\/1995\/01\/01\/patologia-metabolica-de-la-l-carnitina-en-la-hemodialisis\/"},"modified":"1995-01-01T00:00:00","modified_gmt":"1995-01-01T00:00:00","slug":"patologia-metabolica-de-la-l-carnitina-en-la-hemodialisis","status":"publish","type":"post","link":"https:\/\/www.deberes.net\/tesis\/ciencias-medicas\/patologia-metabolica-de-la-l-carnitina-en-la-hemodialisis\/","title":{"rendered":"Patolog\u00eda metabolica de la l-carnitina en la hemodialisis."},"content":{"rendered":"<h2>Tesis doctoral de <strong>  Reyes Amo Belen De Los <\/strong><\/h2>\n<p>La l-carnitina forma parte del mecanismo por el cual los acidos grasos de larga cadena entran en el interior de la mitocondria para su posterior oxidacion. Durante el proceso de la hemodialisis se producen diferentes alteraciones lipidicas y eritrocitarias entre otras, junto con perdidas de carnitina a traves de las membranas de dialisis. El objeto de esta tesis es la de estudiar esas alteraciones y analizar el papel que un tratamiento con l-carnitina pueda ejercer sobre los pacientes. Los pacientes tratados sufrieron una disminucion de los niveles de trialiceridos, y aumentaron los de hdl-colesterol solo en aquellos que presentaron hiper- trigliceridemia. Los niveles de actividad carnitin palmitoiltransferasa eritrocitaria se incrementan en los pacientes tratados, correlacionandose con la disminucion de la relacion acil-carnitina\/carnitina libre. En estos pacientes tambien disminuyo los niveles de reactantes del acido tiobarbiturico, indicadores de lipoperoxidacion, aunque no llegaron a valores controles.<\/p>\n<p>&nbsp;<\/p>\n<h3>Datos acad\u00e9micos de la tesis doctoral \u00ab<strong>Patolog\u00eda metabolica de la l-carnitina en la hemodialisis.<\/strong>\u00ab<\/h3>\n<ul>\n<li><strong>T\u00edtulo de la tesis:<\/strong>\u00a0 Patolog\u00eda metabolica de la l-carnitina en la hemodialisis. <\/li>\n<li><strong>Autor:<\/strong>\u00a0  Reyes Amo Belen De Los <\/li>\n<li><strong>Universidad:<\/strong>\u00a0 Aut\u00f3noma de Madrid<\/li>\n<li><strong>Fecha de lectura de la tesis:<\/strong>\u00a0 01\/01\/1995<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n<h3>Direcci\u00f3n y tribunal<\/h3>\n<ul>\n<li><strong>Director de la tesis<\/strong>\n<ul>\n<li>Joaquin Arenas Barbero<\/li>\n<\/ul>\n<\/li>\n<li><strong>Tribunal<\/strong>\n<ul>\n<li>Presidente del tribunal: Magdalena Ugarte <\/li>\n<li>Florinda Gilsanz Rodriguez (vocal)<\/li>\n<li>\u00e1ngel Del Palacio P\u00e9rez-medel (vocal)<\/li>\n<li>Alberto Fernandez Martinez (vocal)<\/li>\n<\/ul>\n<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n","protected":false},"excerpt":{"rendered":"<p>Tesis doctoral de Reyes Amo Belen De Los La l-carnitina forma parte del mecanismo por el cual los acidos grasos [&hellip;]<\/p>\n","protected":false},"author":1,"featured_media":0,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"site-sidebar-layout":"default","site-content-layout":"","ast-site-content-layout":"","site-content-style":"default","site-sidebar-style":"default","ast-global-header-display":"","ast-banner-title-visibility":"","ast-main-header-display":"","ast-hfb-above-header-display":"","ast-hfb-below-header-display":"","ast-hfb-mobile-header-display":"","site-post-title":"","ast-breadcrumbs-content":"","ast-featured-img":"","footer-sml-layout":"","theme-transparent-header-meta":"","adv-header-id-meta":"","stick-header-meta":"","header-above-stick-meta":"","header-main-stick-meta":"","header-below-stick-meta":"","astra-migrate-meta-layouts":"default","ast-page-background-enabled":"default","ast-page-background-meta":{"desktop":{"background-color":"var(--ast-global-color-4)","background-image":"","background-repeat":"repeat","background-position":"center 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